@article{pop00215,
abstract = {The influence of voltage-gated Ca2+ current (I(Ca)) on Cl- current (I(Cl)) initiated by nicotinic receptors (AChRs) in dialysed voltage-clamped Lymnaea neurons was studied. Depolarising steps applied before or during ACh application decreased I(Cl) transiently and slowed down both the rising phase and decay of I(Cl). The effect of Ica depended on the interval between I(Ca) and I(Cl); it was prevented by intracellular buffering with BAPTA or Ca2+ channel blocking with Ni2+. I(Sr) had a similar action but the recovery was slower than after I(Ca); I(Ba) was ineffective. The data suggest that inactivation of AChR channels is mediated by Ca2+ binding to a site in the AChR or the regulatory protein.},
annote = {Query date: 2020-06-29 13:05:30},
author = {Vulfius, Catherine A. and Ryazansky, Vladimir D. and Krasts, Igor V. and Ilyasov, Fuat E.},
doi = {10.1016/S0304-3940(98)00338-3},
issn = {03043940},
journal = {Neuroscience Letters},
keywords = {Acetylcholine,Cl- current,Intracellular Ca2+,Lymnaea neurons,Nicotinic receptors,Voltage-clamp},
number = {1},
pages = {5--8},
publisher = {Elsevier},
title = {{Intracellular Ca2+ modulates Cl- current evoked by acetylcholine in Lymnaea neurons}},
type = {HTML},
url = {https://www.sciencedirect.com/science/article/pii/S0304394098003383},
volume = {249},
year = {1998}
}

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