Hexabromocyclododecanes and Tetrabromobisphenol-A in Indoor Air and Dust in Birmingham, UK: Implications for Human Exposure. Abdallah, M. A., Harrad, S., & Covaci, A. Environmental Science & Technology, 42(18):6855--6861, September, 2008.
Hexabromocyclododecanes and Tetrabromobisphenol-A in Indoor Air and Dust in Birmingham, UK: Implications for Human Exposure [link]Paper  doi  abstract   bibtex   
Hexabromocyclododecanes (α-, ?-, and ?-HBCDs) and tetrabromobisphenol-A (TBBP-A) were determined in indoor air from homes (n = 33; median concentrations ΣHBCDs = 180 pg m?3; TBBP-A = 15 pg m?3), offices (n = 25; 170; 11), public microenvironments (n = 4; 900; 27) and outdoor air (n = 5; 37; 1). HBCDs and TBBP-A were also determined in dust from homes (n = 45; median concentrations ΣHBCDs = 1300 ng g?1; TBBP-A = 62 ng g?1), offices (n = 28; 760; 36), cars (n = 20; 13,000; 2), and public microenvironments (n = 4; 2700; 230). While ΣHBCDs in car dust significantly exceeded (p \textless 0.05) those in homes and offices, TBBP-A in car dust was significantly lower (p \textless 0.05) than that in homes and offices. No significant differences were observed between ΣHBCDs and TBBP-A in air or dust from homes and offices. Compared to dietary and inhalation exposures, dust ingestion constitutes an important pathway of exposure to HBCDs and TBBP-A for the UK population. Specifically, using average dust ingestion rates and concentrations in dust, dust ingestion constitutes for adults 34% (TBBP-A) and 24% (HBCDs) of overall exposure, and for toddlers 90% (TBBP-A) and 63% (HBCDs). Inhalation appears a minor exposure pathway to both HBCDs and TBBP-A. On average, dust is 33% α-, 11% ?-, and 56% ?-HBCD, while air is 22% α-, 11% ?-, and 65% ?-HBCD.
@article{abdallah_hexabromocyclododecanes_2008,
	title = {Hexabromocyclododecanes and {Tetrabromobisphenol}-{A} in {Indoor} {Air} and {Dust} in {Birmingham}, {UK}: {Implications} for {Human} {Exposure}},
	volume = {42},
	issn = {0013-936X},
	shorttitle = {Hexabromocyclododecanes and {Tetrabromobisphenol}-{A} in {Indoor} {Air} and {Dust} in {Birmingham}, {UK}},
	url = {http://dx.doi.org/10.1021/es801110a},
	doi = {10.1021/es801110a},
	abstract = {Hexabromocyclododecanes (α-, ?-, and ?-HBCDs) and tetrabromobisphenol-A (TBBP-A) were determined in indoor air from homes (n = 33; median concentrations ΣHBCDs = 180 pg m?3; TBBP-A = 15 pg m?3), offices (n = 25; 170; 11), public microenvironments (n = 4; 900; 27) and outdoor air (n = 5; 37; 1). HBCDs and TBBP-A were also determined in dust from homes (n = 45; median concentrations ΣHBCDs = 1300 ng g?1; TBBP-A = 62 ng g?1), offices (n = 28; 760; 36), cars (n = 20; 13,000; 2), and public microenvironments (n = 4; 2700; 230). While ΣHBCDs in car dust significantly exceeded (p {\textless} 0.05) those in homes and offices, TBBP-A in car dust was significantly lower (p {\textless} 0.05) than that in homes and offices. No significant differences were observed between ΣHBCDs and TBBP-A in air or dust from homes and offices. Compared to dietary and inhalation exposures, dust ingestion constitutes an important pathway of exposure to HBCDs and TBBP-A for the UK population. Specifically, using average dust ingestion rates and concentrations in dust, dust ingestion constitutes for adults 34\% (TBBP-A) and 24\% (HBCDs) of overall exposure, and for toddlers 90\% (TBBP-A) and 63\% (HBCDs). Inhalation appears a minor exposure pathway to both HBCDs and TBBP-A. On average, dust is 33\% α-, 11\% ?-, and 56\% ?-HBCD, while air is 22\% α-, 11\% ?-, and 65\% ?-HBCD.},
	number = {18},
	urldate = {2014-09-26TZ},
	journal = {Environmental Science \& Technology},
	author = {Abdallah, Mohamed Abou-Elwafa and Harrad, Stuart and Covaci, Adrian},
	month = sep,
	year = {2008},
	pages = {6855--6861}
}

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