Hexabromocyclododecanes and Tetrabromobisphenol-A in Indoor Air and Dust in Birmingham, UK: Implications for Human Exposure. Abdallah, M. A., Harrad, S., & Covaci, A. Environmental Science & Technology, 42(18):6855–6861, September, 2008. Paper doi abstract bibtex Hexabromocyclododecanes (α-, ?-, and ?-HBCDs) and tetrabromobisphenol-A (TBBP-A) were determined in indoor air from homes (n = 33; median concentrations ΣHBCDs = 180 pg m?3; TBBP-A = 15 pg m?3), offices (n = 25; 170; 11), public microenvironments (n = 4; 900; 27) and outdoor air (n = 5; 37; 1). HBCDs and TBBP-A were also determined in dust from homes (n = 45; median concentrations ΣHBCDs = 1300 ng g?1; TBBP-A = 62 ng g?1), offices (n = 28; 760; 36), cars (n = 20; 13,000; 2), and public microenvironments (n = 4; 2700; 230). While ΣHBCDs in car dust significantly exceeded (p \textless 0.05) those in homes and offices, TBBP-A in car dust was significantly lower (p \textless 0.05) than that in homes and offices. No significant differences were observed between ΣHBCDs and TBBP-A in air or dust from homes and offices. Compared to dietary and inhalation exposures, dust ingestion constitutes an important pathway of exposure to HBCDs and TBBP-A for the UK population. Specifically, using average dust ingestion rates and concentrations in dust, dust ingestion constitutes for adults 34% (TBBP-A) and 24% (HBCDs) of overall exposure, and for toddlers 90% (TBBP-A) and 63% (HBCDs). Inhalation appears a minor exposure pathway to both HBCDs and TBBP-A. On average, dust is 33% α-, 11% ?-, and 56% ?-HBCD, while air is 22% α-, 11% ?-, and 65% ?-HBCD.
@article{abdallah_hexabromocyclododecanes_2008,
title = {Hexabromocyclododecanes and {Tetrabromobisphenol}-{A} in {Indoor} {Air} and {Dust} in {Birmingham}, {UK}: {Implications} for {Human} {Exposure}},
volume = {42},
issn = {0013-936X},
shorttitle = {Hexabromocyclododecanes and {Tetrabromobisphenol}-{A} in {Indoor} {Air} and {Dust} in {Birmingham}, {UK}},
url = {http://dx.doi.org/10.1021/es801110a},
doi = {10.1021/es801110a},
abstract = {Hexabromocyclododecanes (α-, ?-, and ?-HBCDs) and tetrabromobisphenol-A (TBBP-A) were determined in indoor air from homes (n = 33; median concentrations ΣHBCDs = 180 pg m?3; TBBP-A = 15 pg m?3), offices (n = 25; 170; 11), public microenvironments (n = 4; 900; 27) and outdoor air (n = 5; 37; 1). HBCDs and TBBP-A were also determined in dust from homes (n = 45; median concentrations ΣHBCDs = 1300 ng g?1; TBBP-A = 62 ng g?1), offices (n = 28; 760; 36), cars (n = 20; 13,000; 2), and public microenvironments (n = 4; 2700; 230). While ΣHBCDs in car dust significantly exceeded (p {\textless} 0.05) those in homes and offices, TBBP-A in car dust was significantly lower (p {\textless} 0.05) than that in homes and offices. No significant differences were observed between ΣHBCDs and TBBP-A in air or dust from homes and offices. Compared to dietary and inhalation exposures, dust ingestion constitutes an important pathway of exposure to HBCDs and TBBP-A for the UK population. Specifically, using average dust ingestion rates and concentrations in dust, dust ingestion constitutes for adults 34\% (TBBP-A) and 24\% (HBCDs) of overall exposure, and for toddlers 90\% (TBBP-A) and 63\% (HBCDs). Inhalation appears a minor exposure pathway to both HBCDs and TBBP-A. On average, dust is 33\% α-, 11\% ?-, and 56\% ?-HBCD, while air is 22\% α-, 11\% ?-, and 65\% ?-HBCD.},
number = {18},
urldate = {2014-09-26},
journal = {Environmental Science \& Technology},
author = {Abdallah, Mohamed Abou-Elwafa and Harrad, Stuart and Covaci, Adrian},
month = sep,
year = {2008},
pages = {6855--6861},
}
Downloads: 0
{"_id":"ruC5fXwPCygtqaB2v","bibbaseid":"abdallah-harrad-covaci-hexabromocyclododecanesandtetrabromobisphenolainindoorairanddustinbirminghamukimplicationsforhumanexposure-2008","author_short":["Abdallah, M. A.","Harrad, S.","Covaci, A."],"bibdata":{"bibtype":"article","type":"article","title":"Hexabromocyclododecanes and Tetrabromobisphenol-A in Indoor Air and Dust in Birmingham, UK: Implications for Human Exposure","volume":"42","issn":"0013-936X","shorttitle":"Hexabromocyclododecanes and Tetrabromobisphenol-A in Indoor Air and Dust in Birmingham, UK","url":"http://dx.doi.org/10.1021/es801110a","doi":"10.1021/es801110a","abstract":"Hexabromocyclododecanes (α-, ?-, and ?-HBCDs) and tetrabromobisphenol-A (TBBP-A) were determined in indoor air from homes (n = 33; median concentrations ΣHBCDs = 180 pg m?3; TBBP-A = 15 pg m?3), offices (n = 25; 170; 11), public microenvironments (n = 4; 900; 27) and outdoor air (n = 5; 37; 1). HBCDs and TBBP-A were also determined in dust from homes (n = 45; median concentrations ΣHBCDs = 1300 ng g?1; TBBP-A = 62 ng g?1), offices (n = 28; 760; 36), cars (n = 20; 13,000; 2), and public microenvironments (n = 4; 2700; 230). While ΣHBCDs in car dust significantly exceeded (p \\textless 0.05) those in homes and offices, TBBP-A in car dust was significantly lower (p \\textless 0.05) than that in homes and offices. No significant differences were observed between ΣHBCDs and TBBP-A in air or dust from homes and offices. Compared to dietary and inhalation exposures, dust ingestion constitutes an important pathway of exposure to HBCDs and TBBP-A for the UK population. Specifically, using average dust ingestion rates and concentrations in dust, dust ingestion constitutes for adults 34% (TBBP-A) and 24% (HBCDs) of overall exposure, and for toddlers 90% (TBBP-A) and 63% (HBCDs). Inhalation appears a minor exposure pathway to both HBCDs and TBBP-A. On average, dust is 33% α-, 11% ?-, and 56% ?-HBCD, while air is 22% α-, 11% ?-, and 65% ?-HBCD.","number":"18","urldate":"2014-09-26","journal":"Environmental Science & Technology","author":[{"propositions":[],"lastnames":["Abdallah"],"firstnames":["Mohamed","Abou-Elwafa"],"suffixes":[]},{"propositions":[],"lastnames":["Harrad"],"firstnames":["Stuart"],"suffixes":[]},{"propositions":[],"lastnames":["Covaci"],"firstnames":["Adrian"],"suffixes":[]}],"month":"September","year":"2008","pages":"6855–6861","bibtex":"@article{abdallah_hexabromocyclododecanes_2008,\n\ttitle = {Hexabromocyclododecanes and {Tetrabromobisphenol}-{A} in {Indoor} {Air} and {Dust} in {Birmingham}, {UK}: {Implications} for {Human} {Exposure}},\n\tvolume = {42},\n\tissn = {0013-936X},\n\tshorttitle = {Hexabromocyclododecanes and {Tetrabromobisphenol}-{A} in {Indoor} {Air} and {Dust} in {Birmingham}, {UK}},\n\turl = {http://dx.doi.org/10.1021/es801110a},\n\tdoi = {10.1021/es801110a},\n\tabstract = {Hexabromocyclododecanes (α-, ?-, and ?-HBCDs) and tetrabromobisphenol-A (TBBP-A) were determined in indoor air from homes (n = 33; median concentrations ΣHBCDs = 180 pg m?3; TBBP-A = 15 pg m?3), offices (n = 25; 170; 11), public microenvironments (n = 4; 900; 27) and outdoor air (n = 5; 37; 1). HBCDs and TBBP-A were also determined in dust from homes (n = 45; median concentrations ΣHBCDs = 1300 ng g?1; TBBP-A = 62 ng g?1), offices (n = 28; 760; 36), cars (n = 20; 13,000; 2), and public microenvironments (n = 4; 2700; 230). While ΣHBCDs in car dust significantly exceeded (p {\\textless} 0.05) those in homes and offices, TBBP-A in car dust was significantly lower (p {\\textless} 0.05) than that in homes and offices. No significant differences were observed between ΣHBCDs and TBBP-A in air or dust from homes and offices. Compared to dietary and inhalation exposures, dust ingestion constitutes an important pathway of exposure to HBCDs and TBBP-A for the UK population. Specifically, using average dust ingestion rates and concentrations in dust, dust ingestion constitutes for adults 34\\% (TBBP-A) and 24\\% (HBCDs) of overall exposure, and for toddlers 90\\% (TBBP-A) and 63\\% (HBCDs). Inhalation appears a minor exposure pathway to both HBCDs and TBBP-A. On average, dust is 33\\% α-, 11\\% ?-, and 56\\% ?-HBCD, while air is 22\\% α-, 11\\% ?-, and 65\\% ?-HBCD.},\n\tnumber = {18},\n\turldate = {2014-09-26},\n\tjournal = {Environmental Science \\& Technology},\n\tauthor = {Abdallah, Mohamed Abou-Elwafa and Harrad, Stuart and Covaci, Adrian},\n\tmonth = sep,\n\tyear = {2008},\n\tpages = {6855--6861},\n}\n\n\n\n","author_short":["Abdallah, M. A.","Harrad, S.","Covaci, A."],"key":"abdallah_hexabromocyclododecanes_2008","id":"abdallah_hexabromocyclododecanes_2008","bibbaseid":"abdallah-harrad-covaci-hexabromocyclododecanesandtetrabromobisphenolainindoorairanddustinbirminghamukimplicationsforhumanexposure-2008","role":"author","urls":{"Paper":"http://dx.doi.org/10.1021/es801110a"},"metadata":{"authorlinks":{}},"html":""},"bibtype":"article","biburl":"http://bibbase.org/zotero/mdedeo","dataSources":["SSfxPwd7XbkRtnZ9f"],"keywords":[],"search_terms":["hexabromocyclododecanes","tetrabromobisphenol","indoor","air","dust","birmingham","implications","human","exposure","abdallah","harrad","covaci"],"title":"Hexabromocyclododecanes and Tetrabromobisphenol-A in Indoor Air and Dust in Birmingham, UK: Implications for Human Exposure","year":2008}