RNase HI depletion strongly potentiates cell killing by rifampicin in Mycobacteria. Al-Zubaidi, A., Cheung, C., Cook, G. M, Taiaroa, G., Mizrahi, V., Lott, J S., & Dawes, S. S Antimicrobial Agents and Chemotherapy, 66(10):10.1128/aac.02091–21, American Society for Microbiology 1752 N St., N.W., Washington, DC, sep, 2022.
RNase HI depletion strongly potentiates cell killing by rifampicin in Mycobacteria [link]Paper  doi  abstract   bibtex   
Multidrug-resistant (MDR) tuberculosis (TB) is defined by the resistance of Mycobacterium tuberculosis, the causative organism, to the first-line antibiotics rifampicin and isoniazid. Mitigating or...
@article{Al-Zubaidi2022,
abstract = {Multidrug-resistant (MDR) tuberculosis (TB) is defined by the resistance of Mycobacterium tuberculosis, the causative organism, to the first-line antibiotics rifampicin and isoniazid. Mitigating or...},
annote = {OA in biorxiv: RNase HI depletion strongly potentiates cell killing by rifampicin in mycobacteria (biorxiv.org)},
author = {Al-Zubaidi, Abeer and Cheung, Chen-Yi and Cook, Gregory M and Taiaroa, George and Mizrahi, Valerie and Lott, J Shaun and Dawes, Stephanie S},
doi = {10.1128/AAC.02091-21},
issn = {0066-4804},
journal = {Antimicrobial Agents and Chemotherapy},
keywords = {OA,R-loop,RNase HI,antibiotic development,antibiotic resistance,antibiotic synergy,fund{\_}not{\_}ack,original,rifampicin},
mendeley-tags = {OA,fund{\_}not{\_}ack,original},
month = {sep},
number = {10},
pages = {10.1128/aac.02091--21},
pmid = {36154174},
publisher = {American Society for Microbiology 1752 N St., N.W., Washington, DC},
title = {{RNase HI depletion strongly potentiates cell killing by rifampicin in Mycobacteria}},
url = {https://journals.asm.org/doi/10.1128/aac.02091-21},
volume = {66},
year = {2022}
}

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