@article{Bunjun2021a,
abstract = {HIV-1 infection substantially increases the risk of developing tuberculosis (TB). Mechanisms such as defects in the Th1 response to Mycobacterium tuberculosis in HIV-infected persons have been widely reported. However, Th1-independent mechanisms also contribute to protection against TB. To identify},
author = {Bunjun, Rubina and Omondi, Fidilia M A and Makatsa, Mohau S and Keeton, Roanne and Wendoh, Jerome M and M{\"{u}}ller, Tracey L and Prentice, Caryn S L and Wilkinson, Robert J and Riou, Catherine and Burgers, Wendy A},
doi = {10.4049/JIMMUNOL.1900984},
file = {:C$\backslash$:/Users/01462563/AppData/Local/Mendeley Ltd./Mendeley Desktop/Downloaded/Bunjun et al. - 2021 - Th22 cells are a major contributor to the mycobacterial CD4 T cell response and are depleted during HIV infection.pdf:pdf},
issn = {0022-1767},
journal = {The Journal of Immunology},
keywords = {fund{\_}ack,original},
mendeley-tags = {fund{\_}ack,original},
month = {sep},
number = {5},
pages = {1239--1249},
pmid = {34389623},
publisher = {American Association of Immunologists},
title = {{Th22 cells are a major contributor to the mycobacterial CD4+ T cell response and are depleted during HIV infection}},
url = {https://www.jimmunol.org/content/207/5/1239 https://www.jimmunol.org/content/207/5/1239.abstract},
volume = {207},
year = {2021}
}

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