@article{cellai_adenosinergic_2018,
	title = {The {Adenosinergic} {Signaling}: {A} {Complex} but {Promising} {Therapeutic} {Target} for {Alzheimer}'s {Disease}},
	volume = {12},
	issn = {1662-4548},
	shorttitle = {The {Adenosinergic} {Signaling}},
	doi = {10.3389/fnins.2018.00520},
	abstract = {Alzheimer's disease (AD) is the most common neurodegenerative disorder in elderly people. AD is characterized by a progressive cognitive decline and it is neuropathologically defined by two hallmarks: extracellular deposits of aggregated β-amyloid (Aβ) peptides and intraneuronal fibrillar aggregates of hyper- and abnormally phosphorylated Tau proteins. AD results from multiple genetic and environmental risk factors. Epidemiological studies reported beneficial effects of caffeine, a non-selective adenosine receptors antagonist. In the present review, we discuss the impact of caffeine and of adenosinergic system modulation on AD, in terms of pathology and therapeutics.},
	language = {eng},
	journal = {Frontiers in Neuroscience},
	author = {Cellai, Lucrezia and Carvalho, Kevin and Faivre, Emilie and Deleau, Aude and Vieau, Didier and Buée, Luc and Blum, David and Mériaux, Céline and Gomez-Murcia, Victoria},
	year = {2018},
	pmid = {30123104},
	pmcid = {PMC6085480},
	keywords = {Alzheimer’s disease, caffeine, memory, adenosine, adenosine receptors},
	pages = {520}
}

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