@article{
 title = {EGF-ERBB signalling: towards the systems level},
 type = {article},
 year = {2006},
 identifiers = {[object Object]},
 keywords = {Animals,Endocytosis,Epidermal Growth Factor,Fee,Humans,Ligands,Models,Molecular,Oncogene Proteins v-erbB,Phosphatidyl,Physiological,Signal Transduction},
 pages = {505-516},
 volume = {7},
 websites = {http://dx.doi.org/10.1038/nrm1962},
 month = {7},
 institution = {Department of Biological Regulation, the Weizmann Institute of Science, 1 Hertzl Street, Rehovot 76100, Israel.},
 id = {48ecd52c-52b9-3193-80ef-a1b29fdc9fe2},
 created = {2012-04-04T15:37:38.000Z},
 file_attached = {false},
 profile_id = {00fd71da-e805-3e9b-bd6d-ce1da66adfce},
 group_id = {65b20e96-92ca-3d76-a4e9-a84ba22c682d},
 last_modified = {2012-04-04T15:58:54.000Z},
 read = {false},
 starred = {false},
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 citation_key = {Citri2006},
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 abstract = {Signalling through the ERBB/HER receptors is intricately involved in human cancer and already serves as a target for several cancer drugs. Because of its inherent complexity, it is useful to envision ERBB signalling as a bow-tie-configured, evolvable network, which shares modularity, redundancy and control circuits with robust biological and engineered systems. Because network fragility is an inevitable trade-off of robustness, systems-level understanding is expected to generate therapeutic opportunities to intercept aberrant network activation.},
 bibtype = {article},
 author = {Citri, Ami and Yarden, Yosef},
 journal = {Nat Rev Mol Cell Biol},
 number = {7}
}

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