@article{fei_novel_2010,
	title = {A {Novel} {Negative} {Fe}-{Deficiency}-{Responsive} {Element} and a {TGGCA}-{Type}-{Like} {FeRE} {Control} the {Expression} of \textit{{FTR1}} in \textit{{Chlamydomonas} reinhardtii}},
	volume = {2010},
	issn = {1110-7243, 1110-7251},
	url = {http://www.hindawi.com/journals/bmri/2010/790247/},
	doi = {10.1155/2010/790247},
	abstract = {We have reported three Fe-deficiency-responsive elements (FEREs),
              FOX1, ATX1,
              and
              FEA1
              , all of which are positive regulatory elements in response to iron deficiency in
              Chlamydomonas reinhardtii
              . Here we describe
              FTR1
              , another iron regulated gene and mutational analysis of its promoter. Our results reveal that the FeREs of
              FTR1
              distinguish itself from other iron response elements by containing both
              negative
              and
              positive
              regulatory regions. In
              FTR1
              , the
              
                −
              
              291/
              
                −
              
              236 region from the transcriptional start site is necessary and sufficient for Fe-deficiency-inducible expression. This region contains two positive FeREs with a TGGCA-like core sequence: the FtrFeRE1 (A
              TGCA
              GGCT) at
              
                −
              
              287/
              
                −
              
              279 and the FtrFeRE2 (AAGCGAT
              TGCCA
              GAGCGC) at
              
                −
              
              253/
              
                −
              
              236. Furthermore, we identified a novel FERE, FtrFeRE3 (AGTAACTGTTAAGCC) localized at
              
                −
              
              319/
              
                −
              
              292, which negatively influences the expression of
              FTR1
              .},
	language = {en},
	urldate = {2021-06-08},
	journal = {Journal of Biomedicine and Biotechnology},
	author = {Fei, Xiaowen and Eriksson, Mats and Li, Yajun and Deng, Xiaodong},
	year = {2010},
	pages = {1--9},
}

{"_id":"cjLXymebB9Q3fXtDg","bibbaseid":"fei-eriksson-li-deng-anovelnegativefedeficiencyresponsiveelementandatggcatypelikeferecontroltheexpressionofiftr1iinichlamydomonasreinhardtiii-2010","author_short":["Fei, X.","Eriksson, M.","Li, Y.","Deng, X."],"bibdata":{"bibtype":"article","type":"article","title":"A Novel Negative Fe-Deficiency-Responsive Element and a TGGCA-Type-Like FeRE Control the Expression of <i>FTR1</i> in <i>Chlamydomonas reinhardtii</i>","volume":"2010","issn":"1110-7243, 1110-7251","url":"http://www.hindawi.com/journals/bmri/2010/790247/","doi":"10.1155/2010/790247","abstract":"We have reported three Fe-deficiency-responsive elements (FEREs), FOX1, ATX1, and FEA1 , all of which are positive regulatory elements in response to iron deficiency in Chlamydomonas reinhardtii . Here we describe FTR1 , another iron regulated gene and mutational analysis of its promoter. Our results reveal that the FeREs of FTR1 distinguish itself from other iron response elements by containing both negative and positive regulatory regions. In FTR1 , the − 291/ − 236 region from the transcriptional start site is necessary and sufficient for Fe-deficiency-inducible expression. This region contains two positive FeREs with a TGGCA-like core sequence: the FtrFeRE1 (A TGCA GGCT) at − 287/ − 279 and the FtrFeRE2 (AAGCGAT TGCCA GAGCGC) at − 253/ − 236. Furthermore, we identified a novel FERE, FtrFeRE3 (AGTAACTGTTAAGCC) localized at − 319/ − 292, which negatively influences the expression of FTR1 .","language":"en","urldate":"2021-06-08","journal":"Journal of Biomedicine and Biotechnology","author":[{"propositions":[],"lastnames":["Fei"],"firstnames":["Xiaowen"],"suffixes":[]},{"propositions":[],"lastnames":["Eriksson"],"firstnames":["Mats"],"suffixes":[]},{"propositions":[],"lastnames":["Li"],"firstnames":["Yajun"],"suffixes":[]},{"propositions":[],"lastnames":["Deng"],"firstnames":["Xiaodong"],"suffixes":[]}],"year":"2010","pages":"1–9","bibtex":"@article{fei_novel_2010,\n\ttitle = {A {Novel} {Negative} {Fe}-{Deficiency}-{Responsive} {Element} and a {TGGCA}-{Type}-{Like} {FeRE} {Control} the {Expression} of \\textit{{FTR1}} in \\textit{{Chlamydomonas} reinhardtii}},\n\tvolume = {2010},\n\tissn = {1110-7243, 1110-7251},\n\turl = {http://www.hindawi.com/journals/bmri/2010/790247/},\n\tdoi = {10.1155/2010/790247},\n\tabstract = {We have reported three Fe-deficiency-responsive elements (FEREs),\n FOX1, ATX1,\n and\n FEA1\n , all of which are positive regulatory elements in response to iron deficiency in\n Chlamydomonas reinhardtii\n . Here we describe\n FTR1\n , another iron regulated gene and mutational analysis of its promoter. Our results reveal that the FeREs of\n FTR1\n distinguish itself from other iron response elements by containing both\n negative\n and\n positive\n regulatory regions. In\n FTR1\n , the\n \n −\n \n 291/\n \n −\n \n 236 region from the transcriptional start site is necessary and sufficient for Fe-deficiency-inducible expression. This region contains two positive FeREs with a TGGCA-like core sequence: the FtrFeRE1 (A\n TGCA\n GGCT) at\n \n −\n \n 287/\n \n −\n \n 279 and the FtrFeRE2 (AAGCGAT\n TGCCA\n GAGCGC) at\n \n −\n \n 253/\n \n −\n \n 236. Furthermore, we identified a novel FERE, FtrFeRE3 (AGTAACTGTTAAGCC) localized at\n \n −\n \n 319/\n \n −\n \n 292, which negatively influences the expression of\n FTR1\n .},\n\tlanguage = {en},\n\turldate = {2021-06-08},\n\tjournal = {Journal of Biomedicine and Biotechnology},\n\tauthor = {Fei, Xiaowen and Eriksson, Mats and Li, Yajun and Deng, Xiaodong},\n\tyear = {2010},\n\tpages = {1--9},\n}\n\n\n\n\n\n\n\n\n\n\n\n\n\n\n\n","author_short":["Fei, X.","Eriksson, M.","Li, Y.","Deng, X."],"key":"fei_novel_2010","id":"fei_novel_2010","bibbaseid":"fei-eriksson-li-deng-anovelnegativefedeficiencyresponsiveelementandatggcatypelikeferecontroltheexpressionofiftr1iinichlamydomonasreinhardtiii-2010","role":"author","urls":{"Paper":"http://www.hindawi.com/journals/bmri/2010/790247/"},"metadata":{"authorlinks":{}}},"bibtype":"article","biburl":"https://bibbase.org/zotero/upscpub","dataSources":["fvfkWcShg3Mybjoog","Tu3jPdZyJF3j547xT","9cGcv2t8pRzC92kzs","3zTPPmKj8BiTcpc6C"],"keywords":[],"search_terms":["novel","negative","deficiency","responsive","element","tggca","type","fere","control","expression","ftr1","chlamydomonas","reinhardtii","fei","eriksson","li","deng"],"title":"A Novel Negative Fe-Deficiency-Responsive Element and a TGGCA-Type-Like FeRE Control the Expression of <i>FTR1</i> in <i>Chlamydomonas reinhardtii</i>","year":2010}