@article{
 title = {Genetics of aging},
 type = {article},
 year = {1997},
 identifiers = {[object Object]},
 keywords = {Aging/*genetics,Alzheimer Disease/genetics,Animals,Apoptosis/genetics,Gene Expression,Humans,Longevity/*genetics,Mutation,Research Support, Non-U.S. Gov't,Research Support, U.S. Gov't, P.H.S.,Risk Factors,Variation (Genetics)},
 pages = {407-411},
 volume = {278},
 websites = {http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=9334291},
 id = {7125e0bf-17e5-32ca-b8ec-f153683043d6},
 created = {2017-06-19T13:45:43.517Z},
 file_attached = {false},
 profile_id = {de68dde1-2ff3-3a4e-a214-ef424d0c7646},
 group_id = {b2078731-0913-33b9-8902-a53629a24e83},
 last_modified = {2017-06-19T13:45:43.632Z},
 read = {false},
 starred = {false},
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 abstract = {The role of genetics in determining life-span is complex and paradoxical. Although the heritability of life-span is relatively minor, some genetic variants significantly modify senescence of mammals and invertebrates, with both positive and negative impacts on age-related disorders and life-spans. In certain examples, the gene variants alter metabolic pathways, which could thereby mediate interactions with nutritional and other environmental factors that influence life-span. Given the relatively minor effect and variable penetrance of genetic risk factors that appear to affect survival and health at advanced ages, life-style and other environmental influences may profoundly modify outcomes of aging.},
 bibtype = {article},
 author = {Finch, C E and Tanzi, R E},
 journal = {Science},
 number = {5337}
}

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