@Article{Goosens2002,
  author   = {Ki A Goosens and Stephen Maren},
  journal  = {Hippocampus},
  title    = {Long-term potentiation as a substrate for memory: {E}vidence from studies of amygdaloid plasticity and {P}avlovian fear conditioning.},
  year     = {2002},
  number   = {5},
  pages    = {592-9},
  volume   = {12},
  abstract = {Recent reports have raised concerns about the ability of long-term
	potentiation (LTP) to account for associative learning and memory.
	In this paper, we review the many mechanistic similarities between
	one form of associative learning, Pavlovian fear conditioning, and
	amygdaloid LTP. We then address many of the criticisms levied against
	LTP within the framework of fear conditioning. We believe that many
	of the apparent discrepancies between LTP and behavior can be generally
	accounted for by a failure to appreciate that learned behavior is
	supported by multiple synapses in an extensive network of brain structures.
	We conclude that LTP remains a viable substrate for memory.},
  keywords = {Amygdala, Animals, Evaluation Studies, Hippocampus, Human, Learning, Long-Term Potentiation, Memory, Models, Neurological, Neural Pathways, Neuronal Plasticity, Support, Non-U.S. Gov't, Synapses, 2-Amino-5-phosphonovalerate, Electric Stimulation, Isomerism, Rats, Receptors, N-Methyl-D-Aspartate, Neurotransmitter, Valine, Visual Perception, Action Potentials, Calcium Signaling, Central Nervous System, Chelating Agents, Conditioning (Psychology), Cyclic AMP-Dependent Protein Kinases, Enzyme Inhibitors, Excitatory Amino Acid Antagonists, Motor Neurons, Neurons, Afferent, Reaction Time, Reflex, Signal Transduction, U.S. Gov't, P.H.S., Synaptic Transmission, Association Learning, Conditioning, Classical, Fear, 12440575},
}

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We then address many of the criticisms levied against LTP within the framework of fear conditioning. We believe that many of the apparent discrepancies between LTP and behavior can be generally accounted for by a failure to appreciate that learned behavior is supported by multiple synapses in an extensive network of brain structures. We conclude that LTP remains a viable substrate for memory.","keywords":"Amygdala, Animals, Evaluation Studies, Hippocampus, Human, Learning, Long-Term Potentiation, Memory, Models, Neurological, Neural Pathways, Neuronal Plasticity, Support, Non-U.S. Gov't, Synapses, 2-Amino-5-phosphonovalerate, Electric Stimulation, Isomerism, Rats, Receptors, N-Methyl-D-Aspartate, Neurotransmitter, Valine, Visual Perception, Action Potentials, Calcium Signaling, Central Nervous System, Chelating Agents, Conditioning (Psychology), Cyclic AMP-Dependent Protein Kinases, Enzyme Inhibitors, Excitatory Amino Acid Antagonists, Motor Neurons, Neurons, Afferent, Reaction Time, Reflex, Signal Transduction, U.S. Gov't, P.H.S., Synaptic Transmission, Association Learning, Conditioning, Classical, Fear, 12440575","bibtex":"@Article{Goosens2002,\n author = {Ki A Goosens and Stephen Maren},\n journal = {Hippocampus},\n title = {Long-term potentiation as a substrate for memory: {E}vidence from studies of amygdaloid plasticity and {P}avlovian fear conditioning.},\n year = {2002},\n number = {5},\n pages = {592-9},\n volume = {12},\n abstract = {Recent reports have raised concerns about the ability of long-term\n\tpotentiation (LTP) to account for associative learning and memory.\n\tIn this paper, we review the many mechanistic similarities between\n\tone form of associative learning, Pavlovian fear conditioning, and\n\tamygdaloid LTP. We then address many of the criticisms levied against\n\tLTP within the framework of fear conditioning. We believe that many\n\tof the apparent discrepancies between LTP and behavior can be generally\n\taccounted for by a failure to appreciate that learned behavior is\n\tsupported by multiple synapses in an extensive network of brain structures.\n\tWe conclude that LTP remains a viable substrate for memory.},\n keywords = {Amygdala, Animals, Evaluation Studies, Hippocampus, Human, Learning, Long-Term Potentiation, Memory, Models, Neurological, Neural Pathways, Neuronal Plasticity, Support, Non-U.S. Gov't, Synapses, 2-Amino-5-phosphonovalerate, Electric Stimulation, Isomerism, Rats, Receptors, N-Methyl-D-Aspartate, Neurotransmitter, Valine, Visual Perception, Action Potentials, Calcium Signaling, Central Nervous System, Chelating Agents, Conditioning (Psychology), Cyclic AMP-Dependent Protein Kinases, Enzyme Inhibitors, Excitatory Amino Acid Antagonists, Motor Neurons, Neurons, Afferent, Reaction Time, Reflex, Signal Transduction, U.S. Gov't, P.H.S., Synaptic Transmission, Association Learning, Conditioning, Classical, Fear, 12440575},\n}\n\n","author_short":["Goosens, K. 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