Eosinophilic esophagitis after esophageal atresia: is there an association? Case presentation and literature review. Gorter, R. R., Heij, H. A., van der Voorn, J. P., & Kneepkens, C. M. F. Journal of Pediatric Surgery, 47(6):e9–13, June, 2012. doi abstract bibtex Eosinophilic esophagitis (EoE) is a relatively new condition resulting in dysphagia or symptoms resembling gastroesophageal reflux disease, symptoms that also are common in patients with a history of esophageal atresia. We present 2 patients with persistent dysphagia after repair of esophageal atresia that was caused by EoE. Although the exact etiology and pathogenesis of EoE remain unclear, it is now generally accepted that it is the result of a T-helper cell 2-type immune response with a crucial role for the eosinophil-specific chemotaxis factor eotaxin 3 and eosinophils. Because there are genetic similarities between esophageal atresia and EoE, we speculate that patients with esophageal atresia are at increased risk for developing EoE.
@article{gorter_eosinophilic_2012,
title = {Eosinophilic esophagitis after esophageal atresia: is there an association? {Case} presentation and literature review},
volume = {47},
issn = {1531-5037},
shorttitle = {Eosinophilic esophagitis after esophageal atresia},
doi = {10.1016/j.jpedsurg.2012.01.079},
abstract = {Eosinophilic esophagitis (EoE) is a relatively new condition resulting in dysphagia or symptoms resembling gastroesophageal reflux disease, symptoms that also are common in patients with a history of esophageal atresia. We present 2 patients with persistent dysphagia after repair of esophageal atresia that was caused by EoE. Although the exact etiology and pathogenesis of EoE remain unclear, it is now generally accepted that it is the result of a T-helper cell 2-type immune response with a crucial role for the eosinophil-specific chemotaxis factor eotaxin 3 and eosinophils. Because there are genetic similarities between esophageal atresia and EoE, we speculate that patients with esophageal atresia are at increased risk for developing EoE.},
language = {eng},
number = {6},
journal = {Journal of Pediatric Surgery},
author = {Gorter, Ramon R. and Heij, Hugo A. and van der Voorn, J. Patrick and Kneepkens, C. M. Frank},
month = jun,
year = {2012},
keywords = {Abnormalities, Multiple, Anal Canal, Anti-Ulcer Agents, Barrett Esophagus, Chemokine CCL26, Chemokines, CC, Child, Combined Modality Therapy, Deglutition Disorders, Disease Susceptibility, Eosinophilia, Esophageal Atresia, Esophagitis, Esophagus, Female, Follow-Up Studies, Food Hypersensitivity, Forkhead Transcription Factors, Fundoplication, Gastroesophageal Reflux, Gene Deletion, Heart Defects, Congenital, Hernia, Hiatal, Humans, Infant, Newborn, Kidney, Limb Deformities, Congenital, Male, Postoperative Complications, Spine, Th2 Cells, Trachea, Tracheoesophageal Fistula},
pages = {e9--13},
}
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