TU alternans, long QTU, and torsade de pointes: clinical and experimental observations. Habbab, M. & El-Sherif, N. Pacing Clin Electrophysiol, 15(6):916--31, 1992.
  author =       "M.A. Habbab and N. El-Sherif",
  title =        "{TU} alternans, long {QTU}, and torsade de pointes:
                 clinical and experimental observations.",
  journal =      "Pacing Clin Electrophysiol",
  year =         "1992",
  volume =       "15",
  number =       "6",
  pages =        "916--31",
  robnote =      "LQTS, No hard copy TU alternans was investigated by
                 MAP recordings in six normal dogs, following the
                 administration of anthopleurin-A (AP-A), a drug shown
                 to delay sodium inactivation and to induce bradycardia
                 dependent long QTU, EADs, and TdP. Tachycardia
                 dependent TdP in clinical and experimental examples of
                 long QTU seems to be characteristically associated with
                 TU alternans. Dispersion of repolarization may underlie
                 the increased ventricular electrical instability in
                 these cases. Seems to look at EADs and heterogeneous
                 repolarization as causes of TdP in this animal model.
                 Later papers (see Chi98) provide a more thorough
                 examination and mechanism",

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