TU alternans, long QTU, and torsade de pointes: clinical and experimental observations. Habbab, M. & El-Sherif, N. Pacing Clin Electrophysiol, 15(6):916–31, 1992. bibtex @Article{RSM:Hab92,
author = "M.A. Habbab and N. El-Sherif",
title = "{TU} alternans, long {QTU}, and torsade de pointes:
clinical and experimental observations.",
journal = "Pacing Clin Electrophysiol",
year = "1992",
volume = "15",
number = "6",
pages = "916--31",
robnote = "LQTS, No hard copy TU alternans was investigated by
MAP recordings in six normal dogs, following the
administration of anthopleurin-A (AP-A), a drug shown
to delay sodium inactivation and to induce bradycardia
dependent long QTU, EADs, and TdP. Tachycardia
dependent TdP in clinical and experimental examples of
long QTU seems to be characteristically associated with
TU alternans. Dispersion of repolarization may underlie
the increased ventricular electrical instability in
these cases. Seems to look at EADs and heterogeneous
repolarization as causes of TdP in this animal model.
Later papers (see Chi98) provide a more thorough
examination and mechanism",
}
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