@article{kelly_poz_2006,
	title = {{POZ} for effect – {POZ}-{ZF} transcription factors in cancer and development},
	volume = {16},
	issn = {09628924},
	url = {http://www.ncbi.nlm.nih.gov/pubmed/16996269 https://linkinghub.elsevier.com/retrieve/pii/S096289240600239X},
	doi = {10.1016/j.tcb.2006.09.003},
	abstract = {The BTB/POZ-ZF [Broad complex, Tramtrack, Bric à brac (BTB) or poxvirus and zinc finger (POZ)-zinc finger] protein family comprises a diverse group of transcription factors. POZ-ZF proteins have been implicated in many biological processes, including B cell fate determination, DNA damage responses, cell cycle progression and a multitude of developmental events, including gastrulation, limb formation and hematopoietic stem cell fate determination. Consequently, dysfunction of vertebrate POZ-ZF proteins, such as promyelocytic leukemia zinc finger (PLZF), B cell lymphoma 6 (Bcl-6), hypermethylated in cancer 1 (HIC-1), Kaiso, ZBTB7 and Fanconi anemia zinc finger (FAZF), has been linked directly or indirectly to tumorigenesis and developmental disorders. Here, we discuss recent advances in the POZ-ZF field and the implications for the design of future studies to elucidate the biological roles of these unique transcription factors.},
	number = {11},
	journal = {Trends in Cell Biology},
	author = {Kelly, Kevin F. and Daniel, Juliet M.},
	month = nov,
	year = {2006},
	pmid = {16996269},
	pages = {578--587},
}

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