@ARTICLE{Khromykh2004847,
author={Khromykh, Yu.M. and Varentsova, E.R. and Sarantseva, S.V. and Kotlovanova, L.V.},
title={New characteristic of Drosophila rad(2)201G1 mutation: Epigenetic inheritance of repair defect through meiosis and connection with Rad51C gene},
journal={Doklady Akademii Nauk},
year={2004},
volume={395},
number={6},
pages={847-849},
note={cited By 0},
url={https://www.scopus.com/inward/record.uri?eid=2-s2.0-8644274865&partnerID=40&md5=9e209ae072a359c28bfc256ddb461750},
affiliation={Peterb. Inst. Yad. Fiz., Sankt-Peterburg, Russian Federation},
abstract={The effect of rad(2)201G1 mutation on DNA reparation in Drosophila has been studied. Experimental system of mobilization of P-elements with P-transposase has been used as a model. The existence of inheritable mechanism of gene regulation in DNA reparation control has been demonstrated. Reparation deficiency has been probably meiosis-mediated. The rad201 appeared to be a mutant allele of Rad51C gene.},
correspondence_address1={Khromykh, Yu.M.; Peterb. Inst. Yad. Fiz., Sankt-Peterburg, Russian Federation},
issn={08695652},
coden={DAKNE},
language={Russian},
abbrev_source_title={Dokl Akad Nauk},
document_type={Article},
source={Scopus},
}

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