Multifunctional regulatory mutation in Bacillus subtilis flavinogenesis system. Kreneva, R., Karelov, D., Korol'kova, N., Mironov, A., & Perumov, D. Genetika, 45(10):1420-1424, 2009. cited By 0
Paper abstract bibtex Among Bacillus subtilis riboflavin-resistant mutants we identified one, which differed from other regulatory mutants by overproduction of riboflavin and simultaneous upregulation of the rib C gene encoding flavokinase/FAD-synthase. Genetic and biochemical analysis showed that the ribU1 mutation determines a trans-acting factor that simultaneously regulates activity of riboflavin and truB-ribC-rpsO operons. Regulatory activity of the ribU1 mutation comprises about 10% of Rfn element activity on interaction with flavins. The ribUl mutation can be presumably ascribed to a gene of the transcriptional regulators family.
@ARTICLE{Kreneva20091420,
author={Kreneva, R.A. and Karelov, D.V. and Korol'kova, N.V. and Mironov, A.S. and Perumov, D.A.},
title={Multifunctional regulatory mutation in Bacillus subtilis flavinogenesis system},
journal={Genetika},
year={2009},
volume={45},
number={10},
pages={1420-1424},
note={cited By 0},
url={https://www.scopus.com/inward/record.uri?eid=2-s2.0-73349140528&partnerID=40&md5=4f353fcfe7668542224696b82cff7ad3},
abstract={Among Bacillus subtilis riboflavin-resistant mutants we identified one, which differed from other regulatory mutants by overproduction of riboflavin and simultaneous upregulation of the rib C gene encoding flavokinase/FAD-synthase. Genetic and biochemical analysis showed that the ribU1 mutation determines a trans-acting factor that simultaneously regulates activity of riboflavin and truB-ribC-rpsO operons. Regulatory activity of the ribU1 mutation comprises about 10% of Rfn element activity on interaction with flavins. The ribUl mutation can be presumably ascribed to a gene of the transcriptional regulators family.},
correspondence_address1={Kreneva, R.A.},
issn={00166758},
pubmed_id={19947554},
language={Russian},
abbrev_source_title={Genetika},
document_type={Article},
source={Scopus},
}
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