Vascular Neuro-ophthalmology. Lamirel, C., Newman, N. J., & Biousse, V. Handbook of clinical neurology / edited by P.J. Vinken and G.W. Bruyn, 93:595–611, 2009. ![Vascular Neuro-ophthalmology [link]](https://bibbase.org/img/filetypes/link.svg "link")
Paper doi abstract bibtex Visual difficulties are common in patients with stroke. The clinical presentation varies depending on the type of vessel involved (arteries versus veins), the type of stroke (ischemic or hemorrhagic), the size of the arteries involved (small- versus large-artery disease) and the mechanisms of ischemia (embolic, thrombotic, or hemodynamic). Most ocular syndromes and neuroophthalmologic manifestations of stroke vary based on those characteristics and mechanisms. In ischemic arterial cerebrovascular disease, neuroophthalmic symptoms and signs mostly depend on the size and the territory of the artery involved. The retinal microvascular changes are related to incidents of clinical stroke, stroke mortality, coronary artery disease, cerebral white matter changes detected by MRI, and cerebral atrophy. Both infectious and noninfectious inflammation affecting the central nervous system can produce visual symptoms. Patients with systemic hypertension may develop a severe encephalopathy often associated with reversible visual loss. A variety of other systemic disorders producing strokes associated with ocular abnormalities are discussed. The visual pathways are particularly vulnerable to any decrease in their blood or oxygen supply. The effects of decreased cerebral perfusion on visual function vary mostly depending on the collateral circulation. Visual loss may also result from unilateral or bilateral infarction of the retina or optic nerve. The type of neuro-ophthalmic signs depends mostly on the anatomical location of the lesion. Whereas, Terson syndrome describes retinal, subhyaloid, and vitreal hemorrhage that occurs in the setting of subarachnoid hemorrhage. The chapter also discusses embolism, anterior and posterior large-artery circulation ischemia which is often associated with visual symptoms and signs that may precede a cerebral infarction, and primary and secondary intracerebral hemorrhages that may produce visual field defects and abnormal eye movements.
@article{lamirel_vascular_2009,
title = {Vascular {Neuro}-ophthalmology},
volume = {93},
url = {http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2997492/},
doi = {10.1016/S0072-9752(08)93029-3},
abstract = {Visual difficulties are common in patients with stroke. The clinical presentation varies depending on the type of vessel involved (arteries versus veins), the type of stroke (ischemic or hemorrhagic), the size of the arteries involved (small- versus large-artery disease) and the mechanisms of ischemia (embolic, thrombotic, or hemodynamic). Most ocular syndromes and neuroophthalmologic manifestations of stroke vary based on those characteristics and mechanisms. In ischemic arterial cerebrovascular disease, neuroophthalmic symptoms and signs mostly depend on the size and the territory of the artery involved. The retinal microvascular changes are related to incidents of clinical stroke, stroke mortality, coronary artery disease, cerebral white matter changes detected by MRI, and cerebral atrophy. Both infectious and noninfectious inflammation affecting the central nervous system can produce visual symptoms. Patients with systemic hypertension may develop a severe encephalopathy often associated with reversible visual loss. A variety of other systemic disorders producing strokes associated with ocular abnormalities are discussed. The visual pathways are particularly vulnerable to any decrease in their blood or oxygen supply. The effects of decreased cerebral perfusion on visual function vary mostly depending on the collateral circulation. Visual loss may also result from unilateral or bilateral infarction of the retina or optic nerve. The type of neuro-ophthalmic signs depends mostly on the anatomical location of the lesion. Whereas, Terson syndrome describes retinal, subhyaloid, and vitreal hemorrhage that occurs in the setting of subarachnoid hemorrhage. The chapter also discusses embolism, anterior and posterior large-artery circulation ischemia which is often associated with visual symptoms and signs that may precede a cerebral infarction, and primary and secondary intracerebral hemorrhages that may produce visual field defects and abnormal eye movements.},
language = {en},
urldate = {2016-08-16},
journal = {Handbook of clinical neurology / edited by P.J. Vinken and G.W. Bruyn},
author = {Lamirel, Cédric and Newman, Nancy J. and Biousse, Valérie},
editor = {Vinken, P.J. and Bruyn, G.W.},
year = {2009},
pmid = {18804670},
keywords = {\#duplicates},
pages = {595--611},
}
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