@article{lesch_molecular_2008,
	title = {Molecular genetics of adult {ADHD}: converging evidence from genome-wide association and extended pedigree linkage studies.},
	volume = {115},
	issn = {0300-9564},
	url = {http://www.ncbi.nlm.nih.gov/pubmed/18839057},
	doi = {10.1007/s00702-008-0119-3},
	abstract = {A genome-wide association (GWA) study with pooled DNA in adult attention-deficit/hyperactivity disorder (ADHD) employing approximately 500K SNP markers identifies novel risk genes and reveals remarkable overlap with findings from recent GWA scans in substance use disorders. Comparison with results from our previously reported high-resolution linkage scan in extended pedigrees confirms several chromosomal loci, including 16q23.1-24.3 which also reached genome-wide significance in a recent meta-analysis of seven linkage studies (Zhou et al. in Am J Med Genet Part B, 2008). The findings provide additional support for a common effect of genes coding for cell adhesion molecules (e.g., CDH13, ASTN2) and regulators of synaptic plasticity (e.g., CTNNA2, KALRN) despite the complex multifactorial etiologies of adult ADHD and addiction vulnerability.},
	number = {11},
	urldate = {2015-05-16},
	journal = {Journal of neural transmission (Vienna, Austria : 1996)},
	author = {Lesch, Klaus-Peter and Timmesfeld, Nina and Renner, Tobias J and Halperin, Rebecca and Röser, Christoph and Nguyen, T Trang and Craig, David W and Romanos, Jasmin and Heine, Monika and Meyer, Jobst and Freitag, Christine and Warnke, Andreas and Romanos, Marcel and Schäfer, Helmut and Walitza, Susanne and Reif, Andreas and Stephan, Dietrich A and Jacob, Christian},
	month = nov,
	year = {2008},
	pmid = {18839057},
	keywords = {Adult, Attention Deficit Disorder with Hyperactivity, Attention Deficit Disorder with Hyperactivity: gen, Cell Adhesion Molecules, Chromosomes, Chromosomes: genetics, DNA, DNA: genetics, Female, Genetic Linkage, Genetic Linkage: genetics, Genome, Genome-Wide Association Study, Genotype, Human, Humans, Male, Molecular Biology, Neuronal Plasticity, Neuronal Plasticity: physiology, Polymorphism, Single Nucleotide, Single Nucleotide: genetics},
	pages = {1573--85},
}

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A","Jacob, C."],"bibdata":{"bibtype":"article","type":"article","title":"Molecular genetics of adult ADHD: converging evidence from genome-wide association and extended pedigree linkage studies.","volume":"115","issn":"0300-9564","url":"http://www.ncbi.nlm.nih.gov/pubmed/18839057","doi":"10.1007/s00702-008-0119-3","abstract":"A genome-wide association (GWA) study with pooled DNA in adult attention-deficit/hyperactivity disorder (ADHD) employing approximately 500K SNP markers identifies novel risk genes and reveals remarkable overlap with findings from recent GWA scans in substance use disorders. Comparison with results from our previously reported high-resolution linkage scan in extended pedigrees confirms several chromosomal loci, including 16q23.1-24.3 which also reached genome-wide significance in a recent meta-analysis of seven linkage studies (Zhou et al. in Am J Med Genet Part B, 2008). The findings provide additional support for a common effect of genes coding for cell adhesion molecules (e.g., CDH13, ASTN2) and regulators of synaptic plasticity (e.g., CTNNA2, KALRN) despite the complex multifactorial etiologies of adult ADHD and addiction vulnerability.","number":"11","urldate":"2015-05-16","journal":"Journal of neural transmission (Vienna, Austria : 1996)","author":[{"propositions":[],"lastnames":["Lesch"],"firstnames":["Klaus-Peter"],"suffixes":[]},{"propositions":[],"lastnames":["Timmesfeld"],"firstnames":["Nina"],"suffixes":[]},{"propositions":[],"lastnames":["Renner"],"firstnames":["Tobias","J"],"suffixes":[]},{"propositions":[],"lastnames":["Halperin"],"firstnames":["Rebecca"],"suffixes":[]},{"propositions":[],"lastnames":["Röser"],"firstnames":["Christoph"],"suffixes":[]},{"propositions":[],"lastnames":["Nguyen"],"firstnames":["T","Trang"],"suffixes":[]},{"propositions":[],"lastnames":["Craig"],"firstnames":["David","W"],"suffixes":[]},{"propositions":[],"lastnames":["Romanos"],"firstnames":["Jasmin"],"suffixes":[]},{"propositions":[],"lastnames":["Heine"],"firstnames":["Monika"],"suffixes":[]},{"propositions":[],"lastnames":["Meyer"],"firstnames":["Jobst"],"suffixes":[]},{"propositions":[],"lastnames":["Freitag"],"firstnames":["Christine"],"suffixes":[]},{"propositions":[],"lastnames":["Warnke"],"firstnames":["Andreas"],"suffixes":[]},{"propositions":[],"lastnames":["Romanos"],"firstnames":["Marcel"],"suffixes":[]},{"propositions":[],"lastnames":["Schäfer"],"firstnames":["Helmut"],"suffixes":[]},{"propositions":[],"lastnames":["Walitza"],"firstnames":["Susanne"],"suffixes":[]},{"propositions":[],"lastnames":["Reif"],"firstnames":["Andreas"],"suffixes":[]},{"propositions":[],"lastnames":["Stephan"],"firstnames":["Dietrich","A"],"suffixes":[]},{"propositions":[],"lastnames":["Jacob"],"firstnames":["Christian"],"suffixes":[]}],"month":"November","year":"2008","pmid":"18839057","keywords":"Adult, Attention Deficit Disorder with Hyperactivity, Attention Deficit Disorder with Hyperactivity: gen, Cell Adhesion Molecules, Chromosomes, Chromosomes: genetics, DNA, DNA: genetics, Female, Genetic Linkage, Genetic Linkage: genetics, Genome, Genome-Wide Association Study, Genotype, Human, Humans, Male, Molecular Biology, Neuronal Plasticity, Neuronal Plasticity: physiology, Polymorphism, Single Nucleotide, Single Nucleotide: genetics","pages":"1573–85","bibtex":"@article{lesch_molecular_2008,\n\ttitle = {Molecular genetics of adult {ADHD}: converging evidence from genome-wide association and extended pedigree linkage studies.},\n\tvolume = {115},\n\tissn = {0300-9564},\n\turl = {http://www.ncbi.nlm.nih.gov/pubmed/18839057},\n\tdoi = {10.1007/s00702-008-0119-3},\n\tabstract = {A genome-wide association (GWA) study with pooled DNA in adult attention-deficit/hyperactivity disorder (ADHD) employing approximately 500K SNP markers identifies novel risk genes and reveals remarkable overlap with findings from recent GWA scans in substance use disorders. Comparison with results from our previously reported high-resolution linkage scan in extended pedigrees confirms several chromosomal loci, including 16q23.1-24.3 which also reached genome-wide significance in a recent meta-analysis of seven linkage studies (Zhou et al. in Am J Med Genet Part B, 2008). The findings provide additional support for a common effect of genes coding for cell adhesion molecules (e.g., CDH13, ASTN2) and regulators of synaptic plasticity (e.g., CTNNA2, KALRN) despite the complex multifactorial etiologies of adult ADHD and addiction vulnerability.},\n\tnumber = {11},\n\turldate = {2015-05-16},\n\tjournal = {Journal of neural transmission (Vienna, Austria : 1996)},\n\tauthor = {Lesch, Klaus-Peter and Timmesfeld, Nina and Renner, Tobias J and Halperin, Rebecca and Röser, Christoph and Nguyen, T Trang and Craig, David W and Romanos, Jasmin and Heine, Monika and Meyer, Jobst and Freitag, Christine and Warnke, Andreas and Romanos, Marcel and Schäfer, Helmut and Walitza, Susanne and Reif, Andreas and Stephan, Dietrich A and Jacob, Christian},\n\tmonth = nov,\n\tyear = {2008},\n\tpmid = {18839057},\n\tkeywords = {Adult, Attention Deficit Disorder with Hyperactivity, Attention Deficit Disorder with Hyperactivity: gen, Cell Adhesion Molecules, Chromosomes, Chromosomes: genetics, DNA, DNA: genetics, Female, Genetic Linkage, Genetic Linkage: genetics, Genome, Genome-Wide Association Study, Genotype, Human, Humans, Male, Molecular Biology, Neuronal Plasticity, Neuronal Plasticity: physiology, Polymorphism, Single Nucleotide, Single Nucleotide: genetics},\n\tpages = {1573--85},\n}\n\n","author_short":["Lesch, K.","Timmesfeld, N.","Renner, T. 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