@Article{Lutgens_2002_1366,
  author = {Lutgens, E. and Gijbels, M. and Smook, M. and Heeringa, P. and Gotwals, P. and Koteliansky, V. E. and Daemen, M. J.},
 journal = {Arterioscler.Thromb.Vasc.Biol.},
 note = {DA - 20020617 NOT IN FILE},
 number = {6},
 pages = {975-982},
 title = {Transforming growth factor-beta mediates balance between inflammation and fibrosis during plaque progression},
 volume = {22},
 year = {2002},
 keywords = {administration, &, dosage, Animals, antagonists, &, inhibitors, Apolipoproteins, Apolipoproteins, E, Arteriosclerosis, Atherosclerosis, Carotid, Stenosis, Comparative, Study, deficiency, Disease, Progression, Drug, Administration, Schedule, drug, effects, Fibrin, Fibrosis, genetics, Hemorrhage, Immunoglobulin, Fc, Fragments, Immunoglobulin, G, Inflammation, Male, metabolism, Mice, Mice, Inbred, C57BL, Mice, Mutant, Strains, pathology, pharmacology, Phenotype, physiology, Receptors, Transforming, Growth, Factor, beta, Recombinant, Fusion, Proteins, Research, Support, Non-U.S.Gov't, Signal, Transduction, Solubility, Transforming, Growth, Factor, beta},
 title_with_no_special_chars = {Transforming growth factorbeta mediates balance between inflammation and fibrosis during plaque progression}
}

{"_id":"BzsQosfYSuPAD5eNZ","bibbaseid":"lutgens-gijbels-smook-heeringa-gotwals-koteliansky-daemen-transforminggrowthfactorbetamediatesbalancebetweeninflammationandfibrosisduringplaqueprogression-2002","downloads":0,"creationDate":"2018-01-22T16:01:00.440Z","title":"Transforming growth factor-beta mediates balance between inflammation and fibrosis during plaque progression","author_short":["Lutgens, E.","Gijbels, M.","Smook, M.","Heeringa, P.","Gotwals, P.","Koteliansky, V. E.","Daemen, M. J."],"year":2002,"bibtype":"article","biburl":"http://www.uh.edu/bti/_files/bibsync_export2018-01-19.bib","bibdata":{"bibtype":"article","type":"article","author":[{"propositions":[],"lastnames":["Lutgens"],"firstnames":["E."],"suffixes":[]},{"propositions":[],"lastnames":["Gijbels"],"firstnames":["M."],"suffixes":[]},{"propositions":[],"lastnames":["Smook"],"firstnames":["M."],"suffixes":[]},{"propositions":[],"lastnames":["Heeringa"],"firstnames":["P."],"suffixes":[]},{"propositions":[],"lastnames":["Gotwals"],"firstnames":["P."],"suffixes":[]},{"propositions":[],"lastnames":["Koteliansky"],"firstnames":["V.","E."],"suffixes":[]},{"propositions":[],"lastnames":["Daemen"],"firstnames":["M.","J."],"suffixes":[]}],"journal":"Arterioscler.Thromb.Vasc.Biol.","note":"DA - 20020617 NOT IN FILE","number":"6","pages":"975-982","title":"Transforming growth factor-beta mediates balance between inflammation and fibrosis during plaque progression","volume":"22","year":"2002","keywords":"administration, &, dosage, Animals, antagonists, &, inhibitors, Apolipoproteins, Apolipoproteins, E, Arteriosclerosis, Atherosclerosis, Carotid, Stenosis, Comparative, Study, deficiency, Disease, Progression, Drug, Administration, Schedule, drug, effects, Fibrin, Fibrosis, genetics, Hemorrhage, Immunoglobulin, Fc, Fragments, Immunoglobulin, G, Inflammation, Male, metabolism, Mice, Mice, Inbred, C57BL, Mice, Mutant, Strains, pathology, pharmacology, Phenotype, physiology, Receptors, Transforming, Growth, Factor, beta, Recombinant, Fusion, Proteins, Research, Support, Non-U.S.Gov't, Signal, Transduction, Solubility, Transforming, Growth, Factor, beta","title_with_no_special_chars":"Transforming growth factorbeta mediates balance between inflammation and fibrosis during plaque progression","bibtex":"@Article{Lutgens_2002_1366,\r\n author = {Lutgens, E. and Gijbels, M. and Smook, M. and Heeringa, P. and Gotwals, P. and Koteliansky, V. E. and Daemen, M. J.},\r\n journal = {Arterioscler.Thromb.Vasc.Biol.},\r\n note = {DA - 20020617 NOT IN FILE},\r\n number = {6},\r\n pages = {975-982},\r\n title = {Transforming growth factor-beta mediates balance between inflammation and fibrosis during plaque progression},\r\n volume = {22},\r\n year = {2002},\r\n keywords = {administration, &, dosage, Animals, antagonists, &, inhibitors, Apolipoproteins, Apolipoproteins, E, Arteriosclerosis, Atherosclerosis, Carotid, Stenosis, Comparative, Study, deficiency, Disease, Progression, Drug, Administration, Schedule, drug, effects, Fibrin, Fibrosis, genetics, Hemorrhage, Immunoglobulin, Fc, Fragments, Immunoglobulin, G, Inflammation, Male, metabolism, Mice, Mice, Inbred, C57BL, Mice, Mutant, Strains, pathology, pharmacology, Phenotype, physiology, Receptors, Transforming, Growth, Factor, beta, Recombinant, Fusion, Proteins, Research, Support, Non-U.S.Gov't, Signal, Transduction, Solubility, Transforming, Growth, Factor, beta},\r\n title_with_no_special_chars = {Transforming growth factorbeta mediates balance between inflammation and fibrosis during plaque progression}\r\n}\r\n\r\n","author_short":["Lutgens, E.","Gijbels, M.","Smook, M.","Heeringa, P.","Gotwals, P.","Koteliansky, V. E.","Daemen, M. J."],"key":"Lutgens_2002_1366","id":"Lutgens_2002_1366","bibbaseid":"lutgens-gijbels-smook-heeringa-gotwals-koteliansky-daemen-transforminggrowthfactorbetamediatesbalancebetweeninflammationandfibrosisduringplaqueprogression-2002","role":"author","urls":{},"keyword":["administration","&","dosage","Animals","antagonists","&","inhibitors","Apolipoproteins","Apolipoproteins","E","Arteriosclerosis","Atherosclerosis","Carotid","Stenosis","Comparative","Study","deficiency","Disease","Progression","Drug","Administration","Schedule","drug","effects","Fibrin","Fibrosis","genetics","Hemorrhage","Immunoglobulin","Fc","Fragments","Immunoglobulin","G","Inflammation","Male","metabolism","Mice","Mice","Inbred","C57BL","Mice","Mutant","Strains","pathology","pharmacology","Phenotype","physiology","Receptors","Transforming","Growth","Factor","beta","Recombinant","Fusion","Proteins","Research","Support","Non-U.S.Gov't","Signal","Transduction","Solubility","Transforming","Growth","Factor","beta"],"downloads":0},"search_terms":["transforming","growth","factor","beta","mediates","balance","between","inflammation","fibrosis","during","plaque","progression","lutgens","gijbels","smook","heeringa","gotwals","koteliansky","daemen"],"keywords":["administration","&","dosage","animals","antagonists","&","inhibitors","apolipoproteins","apolipoproteins","e","arteriosclerosis","atherosclerosis","carotid","stenosis","comparative","study","deficiency","disease","progression","drug","administration","schedule","drug","effects","fibrin","fibrosis","genetics","hemorrhage","immunoglobulin","fc","fragments","immunoglobulin","g","inflammation","male","metabolism","mice","mice","inbred","c57bl","mice","mutant","strains","pathology","pharmacology","phenotype","physiology","receptors","transforming","growth","factor","beta","recombinant","fusion","proteins","research","support","non-u.s.gov't","signal","transduction","solubility","transforming","growth","factor","beta"],"authorIDs":[],"dataSources":["9cexBw6hrwgyZphZZ"]}