@article{ohno_apraxic_2000,
	title = {Apraxic agraphia due to thalamic infarction},
	volume = {54},
	doi = {10/grrsrk},
	abstract = {The authors report a patient of pure apraxic agraphia with normal praxis due to left thalamic infarction. 15O-gas-PET showed reduced oxygen metabolism in the left thalamus and the left dorsolateral premotor area, while MRI and 11C-fulumazenil-PET showed no remarkable lesions in the frontal cortex. The patient's word imaging remained normal. The authors hypothesize that thalamic destruction causes pure apraxic agraphia by exerting a remote effect on left dorsolateral premotor area and blocking somewhere between graphemic area and motor programming.},
	number = {12},
	journal = {Neurology},
	author = {Ohno, T. and Bando, M. and Nagura, H. and Ishii, K. and Yamanouchi, H.},
	year = {2000},
	keywords = {\#nosource, Aged, Agraphia/diagnosis/*etiology, Brain Infarction/*complications/diagnosis, Case Report, Human, Language Tests, Magnetic Resonance Imaging, Male, Mediodorsal Thalamic Nucleus/blood supply/*pathology/radionuclide imaging, Neuropsychological Tests, Thalamic Diseases/*complications/diagnosis, Tomography, Emission-Computed},
	pages = {2336--2339},
}

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