Preterm birth and its long-term effects: methylation to mechanisms. Parets, S. E, Bedient, C. E, Menon, R., & Smith, A. K Biology, 3(3):498–513, January, 2014.
Preterm birth and its long-term effects: methylation to mechanisms. [link]Paper  doi  abstract   bibtex   
The epigenetic patterns established during development may influence gene expression over a lifetime and increase susceptibility to chronic disease. Being born preterm (\textless37 weeks of gestation) is associated with increased risk mortality and morbidity from birth until adulthood. This brief review explores the potential role of DNA methylation in preterm birth (PTB) and its possible long-term consequences and provides an overview of the physiological processes central to PTB and recent DNA methylation studies of PTB.
@article{parets_preterm_2014,
	title = {Preterm birth and its long-term effects: methylation to mechanisms.},
	volume = {3},
	issn = {2079-7737},
	url = {http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=4192624&tool=pmcentrez&rendertype=abstract},
	doi = {10.3390/biology3030498},
	abstract = {The epigenetic patterns established during development may influence gene expression over a lifetime and increase susceptibility to chronic disease. Being born preterm ({\textless}37 weeks of gestation) is associated with increased risk mortality and morbidity from birth until adulthood. This brief review explores the potential role of DNA methylation in preterm birth (PTB) and its possible long-term consequences and provides an overview of the physiological processes central to PTB and recent DNA methylation studies of PTB.},
	number = {3},
	urldate = {2015-05-26},
	journal = {Biology},
	author = {Parets, Sasha E and Bedient, Carrie E and Menon, Ramkumar and Smith, Alicia K},
	month = jan,
	year = {2014},
	pmid = {25256426},
	pages = {498--513},
}

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