Altered miRNA expression patterns in Tff2 knock-out mice correlate with cellular pathways of neoplastic development and caloric metabolism. Shah, A. A., Leidinger, P., Keller, A., Wendschlag, A., Meese, E., & Blin, N. International journal of molecular medicine, 29:637–643, April, 2012.
doi  abstract   bibtex   
The trefoil peptide family, consisting in mammals of three members namely TFF1, 2 and 3, plays a cytoprotective role in epithelial cells of various tissues, mainly in the digestive tract. Tff1, Tff2 or Tff3 knock-out mouse models developed various kinds of gastrointestinal impairment. microRNAs are known to be novel gene regulators. We aimed to investigate the physiological role of such miRNAs in Tff2 knock-out mice. Whole miRNome profiling and in silico analysis were performed for Tff2-KO and WT mice. Our latest data explored the role of miRNAs in the regulatory cascades and molecular processes of Tff2-/- mice. As much as 6% of the Tff2-KO mice miRNome was significantly dys-regulated. Further in silico analysis suggests that the respective dys-regulated part of the miRNome is involved in human pathological processes, including pancreatic, colorectal and basal cell cancer. Additionally, the dys-regulated miRNome targets pathways involved in carbohydrate metabolism and adipocytokine signaling. The latter links deficient caloric maintenance in Tff2 and previous observation in Tff3-KO mice with miRNAs. In summary, our proof-of-concept study indicates that miRNAs may play an important role in the regulatory processes of the trefoil peptide family, especially in the regulation of cancer-related cascades.
@Article{Shah2012,
  author          = {Shah, Aftab Ali and Leidinger, Petra and Keller, Andreas and Wendschlag, Anke and Meese, Eckart and Blin, Nikolaus},
  title           = {Altered miRNA expression patterns in Tff2 knock-out mice correlate with cellular pathways of neoplastic development and caloric metabolism.},
  journal         = {International journal of molecular medicine},
  year            = {2012},
  volume          = {29},
  pages           = {637--643},
  month           = apr,
  issn            = {1791-244X},
  abstract        = {The trefoil peptide family, consisting in mammals of three members namely TFF1, 2 and 3, plays a cytoprotective role in epithelial cells of various tissues, mainly in the digestive tract. Tff1, Tff2 or Tff3 knock-out mouse models developed various kinds of gastrointestinal impairment. microRNAs are known to be novel gene regulators. We aimed to investigate the physiological role of such miRNAs in Tff2 knock-out mice. Whole miRNome profiling and in silico analysis were performed for Tff2-KO and WT mice. Our latest data explored the role of miRNAs in the regulatory cascades and molecular processes of Tff2-/- mice. As much as 6% of the Tff2-KO mice miRNome was significantly dys-regulated. Further in silico analysis suggests that the respective dys-regulated part of the miRNome is involved in human pathological processes, including pancreatic, colorectal and basal cell cancer. Additionally, the dys-regulated miRNome targets pathways involved in carbohydrate metabolism and adipocytokine signaling. The latter links deficient caloric maintenance in Tff2 and previous observation in Tff3-KO mice with miRNAs. In summary, our proof-of-concept study indicates that miRNAs may play an important role in the regulatory processes of the trefoil peptide family, especially in the regulation of cancer-related cascades.},
  chemicals       = {Adipokines, MicroRNAs, Mucins, Muscle Proteins, Peptides, TFF2 protein, human, TFF2 protein, mouse, Trefoil Factor-2},
  citation-subset = {IM},
  completed       = {2012-05-29},
  country         = {Greece},
  doi             = {10.3892/ijmm.2012.881},
  issn-linking    = {1107-3756},
  issue           = {4},
  keywords        = {Adipokines, genetics, metabolism; Animals; Carbohydrate Metabolism, genetics; Gene Expression Profiling; Gene Expression Regulation, Neoplastic; Genes, Regulator; Mice; Mice, Knockout; MicroRNAs, genetics, metabolism; Mucins, genetics, metabolism; Muscle Proteins, genetics, metabolism; Peptides, genetics, metabolism; Signal Transduction; Trefoil Factor-2},
  nlm-id          = {9810955},
  owner           = {NLM},
  pmc             = {PMC3573770},
  pmid            = {22245972},
  pubmodel        = {Print-Electronic},
  pubstatus       = {ppublish},
  revised         = {2016-11-25},
}

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