Automated population of Cyc: extracting information about named-entities from the web. Shah, P., Schneider, D., Matuszek, C., Kahlert, R., C., Aldag, B., Baxter, D., Cabral, J., Witbrock, M., & Curtis, J. In Proceedings of the Nineteenth International FLAIRS Conference, volume 141, pages 153-158, 2006. AAAI Press.
Automated population of Cyc: extracting information about named-entities from the web [pdf]Website  abstract   bibtex   
There is persuasive clinical and experimental evidence that macrophages promote cancer initiation and malignant progression. During tumor initiation, they create an inflammatory environment that is mutagenic and promotes growth. As tumors progress to malignancy, macrophages stimulate angiogenesis, enhance tumor cell migration and invasion, and suppress antitumor immunity. At metastatic sites, macrophages prepare the target tissue for arrival of tumor cells, and then a different subpopulation of macrophages promotes tumor cell extravasation, survival, and subsequent growth. Specialized subpopulations of macrophages may represent important new therapeutic targets.

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