@article{
 title = {Role of NMDA receptor GluN2D subunit in the antidepressant effects of enantiomers of ketamine},
 type = {article},
 year = {2017},
 identifiers = {[object Object]},
 websites = {https://www.sciencedirect.com/science/article/pii/S134786131730186X},
 month = {11},
 publisher = {Elsevier},
 day = {10},
 id = {0a0b47eb-ad1a-39b4-a7a3-07e0c51161b8},
 created = {2017-12-19T15:00:44.396Z},
 accessed = {2017-11-27},
 file_attached = {false},
 profile_id = {38c6dbcb-2394-3f18-9217-58d777c08c69},
 group_id = {d9389c6c-8ab5-3b8b-86ed-33db09ca0198},
 last_modified = {2017-12-27T00:58:48.969Z},
 tags = {OA},
 read = {false},
 starred = {false},
 authored = {false},
 confirmed = {true},
 hidden = {false},
 citation_key = {SoichiroIdeYuikoIkekuboaMasayoshiMishinaKenjiHashimoto2017b},
 private_publication = {false},
 abstract = {We investigated the rapid and sustained antidepressant effects of enantiomers of ketamine in N-methyl-d-aspartate (NMDA) receptor GluN2D subunit knockout (GluN2D-KO) mice. Intraperitoneal administration of ketamine or its enantiomers 10 min before the tail-suspension test exerted significant antidepressant effects on restraint stress-induced depression in both wildtype and GluN2D-KO mice. The antidepressant effects of (RS)-ketamine and (S)-ketamine were sustained 96 h after the injection in both wildtype and GluN2D-KO mice, but such sustained antidepressant effects of (R)-ketamine were only observed in wildtype mice. These data suggest that the GluN2D subunit is critical for the sustained antidepressant effects of (R)-ketamine.},
 bibtype = {article},
 author = {Soichiro Ide, Yuiko Ikekuboa, Masayoshi Mishina, Kenji Hashimoto, Kazutaka Ikeda},
 journal = {Journal of Pharmacological Sciences}
}

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