CRISPR/Cas9-mediated disruption of SHANK3 in monkey leads to drug-treatable autism-like symptoms

CRISPR/Cas9-mediated disruption of SHANK3 in monkey leads to drug-treatable autism-like symptoms. Tu, Z., Zhao, H., Li, B., Yan, S., Wang, L., Tang, Y., Li, Z., Bai, D., Li, C., Lin, Y., Li, Y., Liu, J., Xu, H., Guo, X., Jiang, Y., Zhang, Y. Q., & Li, X. Human Molecular Genetics.

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Abstract. Monogenic mutations in the SHANK3 gene, which encodes a postsynaptic scaffold protein, play a causative role in autism spectrum disorder (ASD). Altho

@article{tu_crispr/cas9-mediated_nodate,
	title = {{CRISPR}/{Cas9}-mediated disruption of {SHANK3} in monkey leads to drug-treatable autism-like symptoms},
	url = {https://academic.oup.com/hmg/advance-article/doi/10.1093/hmg/ddy367/5133462},
	doi = {10.1093/hmg/ddy367},
	abstract = {Abstract.  Monogenic mutations in the SHANK3 gene, which encodes a postsynaptic scaffold protein, play a causative role in autism spectrum disorder (ASD). Altho},
	language = {en},
	urldate = {2018-10-29},
	journal = {Human Molecular Genetics},
	author = {Tu, Zhuchi and Zhao, Hui and Li, Bang and Yan, Sen and Wang, Lu and Tang, Yongjin and Li, Zhujun and Bai, Dazhang and Li, Caijuan and Lin, Yinqi and Li, Yuefeng and Liu, Jianrong and Xu, Hao and Guo, Xiangyu and Jiang, Yong-hui and Zhang, Yong Q. and Li, Xiao-Jiang},
}

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