{"_id":"rJ9hfnQN3bescxmMp","bibbaseid":"yu-zhang-zhao-chang-wei-sun-jiang-jiang-etal-cholecystokinintypebreceptormediatedinhibitionofatypekchannelsenhancessensoryneuronalexcitabilitythroughthephosphatidylinositol3kinaseandcsrcdependentjnkpathway-2019","author_short":["Yu, S.","Zhang, Y.","Zhao, X.","Chang, Z.","Wei, Y.","Sun, Y.","Jiang, D.","Jiang, X.","Tao, J."],"bibdata":{"bibtype":"article","type":"article","title":"Cholecystokinin type B receptor-mediated inhibition of A-type K(+) channels enhances sensory neuronal excitability through the phosphatidylinositol 3-kinase and c-Src-dependent JNK pathway.","volume":"17","issn":"1478-811X","doi":"10.1186/s12964-019-0385-8","language":"eng","number":"1","journal":"Cell Communication and Signaling","author":[{"propositions":[],"lastnames":["Yu"],"firstnames":["Shumin"],"suffixes":[]},{"propositions":[],"lastnames":["Zhang"],"firstnames":["Yuan"],"suffixes":[]},{"propositions":[],"lastnames":["Zhao"],"firstnames":["Xianyang"],"suffixes":[]},{"propositions":[],"lastnames":["Chang"],"firstnames":["Zhigang"],"suffixes":[]},{"propositions":[],"lastnames":["Wei"],"firstnames":["Yuan"],"suffixes":[]},{"propositions":[],"lastnames":["Sun"],"firstnames":["Yufang"],"suffixes":[]},{"propositions":[],"lastnames":["Jiang"],"firstnames":["Dongsheng"],"suffixes":[]},{"propositions":[],"lastnames":["Jiang"],"firstnames":["Xinghong"],"suffixes":[]},{"propositions":[],"lastnames":["Tao"],"firstnames":["Jin"],"suffixes":[]}],"month":"June","year":"2019","note":"PMC6582535","keywords":"*A-type K+ channel, *Cholecystokinin, *Dorsal root ganglion, *Neuronal excitability, Animals, CSK Tyrosine-Protein Kinase/*metabolism, Ganglia, Spinal/cytology, MAP Kinase Signaling System/*drug effects, Male, Mice, Mice, Inbred ICR, Nociception/drug effects, Pain/pathology/physiopathology, Phosphatidylinositol 3-Kinase/*metabolism, Potassium Channel Blockers/*pharmacology, Receptor, Cholecystokinin B/*metabolism, Sensory Receptor Cells/*cytology/*drug effects, Sincalide/pharmacology","pages":"68","author_short":["Yu, S.","Zhang, Y.","Zhao, X.","Chang, Z.","Wei, Y.","Sun, Y.","Jiang, D.","Jiang, X.","Tao, J."],"key":"yu_cholecystokinin_2019","id":"yu_cholecystokinin_2019","bibbaseid":"yu-zhang-zhao-chang-wei-sun-jiang-jiang-etal-cholecystokinintypebreceptormediatedinhibitionofatypekchannelsenhancessensoryneuronalexcitabilitythroughthephosphatidylinositol3kinaseandcsrcdependentjnkpathway-2019","role":"author","urls":{},"keyword":["*A-type K+ channel","*Cholecystokinin","*Dorsal root ganglion","*Neuronal excitability","Animals","CSK Tyrosine-Protein Kinase/*metabolism","Ganglia","Spinal/cytology","MAP Kinase Signaling System/*drug effects","Male","Mice","Mice","Inbred ICR","Nociception/drug effects","Pain/pathology/physiopathology","Phosphatidylinositol 3-Kinase/*metabolism","Potassium Channel Blockers/*pharmacology","Receptor","Cholecystokinin B/*metabolism","Sensory Receptor Cells/*cytology/*drug effects","Sincalide/pharmacology"],"metadata":{"authorlinks":{}},"html":""},"bibtype":"article","biburl":"https://bibbase.org/zotero/SPoulson","dataSources":["pwvGX3fpM88G5WEck","KtJiSodd5GdtNoL2N"],"keywords":["*a-type k+ channel","*cholecystokinin","*dorsal root ganglion","*neuronal excitability","animals","csk tyrosine-protein kinase/*metabolism","ganglia","spinal/cytology","map kinase signaling system/*drug effects","male","mice","mice","inbred icr","nociception/drug effects","pain/pathology/physiopathology","phosphatidylinositol 3-kinase/*metabolism","potassium channel blockers/*pharmacology","receptor","cholecystokinin b/*metabolism","sensory receptor cells/*cytology/*drug effects","sincalide/pharmacology"],"search_terms":["cholecystokinin","type","receptor","mediated","inhibition","type","channels","enhances","sensory","neuronal","excitability","through","phosphatidylinositol","kinase","src","dependent","jnk","pathway","yu","zhang","zhao","chang","wei","sun","jiang","jiang","tao"],"title":"Cholecystokinin type B receptor-mediated inhibition of A-type K(+) channels enhances sensory neuronal excitability through the phosphatidylinositol 3-kinase and c-Src-dependent JNK pathway.","year":2019}